Biochemistry of signal transduction and regulation
/ Gerhard Krauss
Biochemistry of signal transduction and regulation
Author
/ Gerhard KraussEdition 1
list
شناسنامه
Main Entry
Krauss, Gerhard، نویسندهTitle and Author
Biochemistry of signal transduction and regulation / / Gerhard KraussPublication Details
، 2003مPhysical Description
xvi, 541 p.؛ :ill. (some col.).Subject
Cellular signal transductionCellular control mechanisms
Language
انگلیسیRetrieval Number
Congress:
Cataloger
کانورتورPrevious ed.: 2001.
Includes bibliographical references and index.
The regulation of gene expression. -- The regulation of enzyme activity. -- Structure and function of signal pathways. -- Signaling by nuclear receptors. -- G protein-coupled signal transmission pathways. -- Intracellular messenger substance: second messengers. -- Ser/Thr-specific protein kinases and protein phosphates. -- Signal transmission via transmembrane receptors with tyrosine-specific protein kinase activity. -- Signal transmission via ras proteins. -- Intracellular signal transduction: the protein cascades of the MAP kinase pathways. -- Membrane receptors with associated tyrosine kinase activity. -- Other receptor classes. -- Regulation of the cell cycle. -- Malfunction of signaling pathways and tumorigenesis: oncogenes and tumor suppressor genes. -- Apoptosis.
Includes bibliographical references and index.
The regulation of gene expression. -- The regulation of enzyme activity. -- Structure and function of signal pathways. -- Signaling by nuclear receptors. -- G protein-coupled signal transmission pathways. -- Intracellular messenger substance: second messengers. -- Ser/Thr-specific protein kinases and protein phosphates. -- Signal transmission via transmembrane receptors with tyrosine-specific protein kinase activity. -- Signal transmission via ras proteins. -- Intracellular signal transduction: the protein cascades of the MAP kinase pathways. -- Membrane receptors with associated tyrosine kinase activity. -- Other receptor classes. -- Regulation of the cell cycle. -- Malfunction of signaling pathways and tumorigenesis: oncogenes and tumor suppressor genes. -- Apoptosis.
